Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. On the other hand, our power to rescue SERT purpose in KI midbrain synaptoneurosomes with the inhibition of FAK implies elevated FAK signaling downstream with the Pro32Pro33 mutant, as confirmed by elevated pFAK localization in five-HT https://pro33-login89888.full-design.com/pro33-an-overview-74893216